Raghu Kalluri

Division of Matrix Biology, Harvard Medical School, United States of America

Raghu Kalluri

Academic positions:
- Associate Professor, Department of Medicine, Beth Israel Deaconess Medical Center
- Director of the Center for Matrix Biology, Beth Israel Deaconess Medical Center.
- Associate Professor of Medicine - Harvard Medical School
- Faculty Member, Biomedical and Biological Sciences (BBS) Graduate Program at the Harvard Medical School
- Faculty, MIT-Harvard Division of Health Sciences and Technology

All industry positions (in last five years):
None declared

Research interests:
The central focus of the laboratory is the study of cellular microenvironment, as determined by extra cellular matrix (ECM) and basement membranes (BM), in the regulation of the cellular behavior during health and disease. This fundamental research interest in matrix biology, coupled with teaching at the Medical School constitutes bulk of my academic activity at the Beth Israel Deaconess Medical Center and Harvard Medical School. The research activities are predominantly in the areas of Vascular Biology and Angiogenesis, Genetic and Acquired Kidney Diseases, Organ Fibrosis and Basement Membrane assembly as it relates to Stem Cell Research and Tissue Engineering.

Any other information:
Professional Societies:
1995- American Society of Nephrology
1999- American Association for the Advancement of Science
2003- American Association of Cancer Research

Any competing interests declared are displayed with individual evaluations.

: Nephrology > Pathophysiology of Chronic Kidney Disease (CKD) (15 November 2005 - 30 September 2008)

Links

http://www.hms.harvard.edu/dms/bbs/fac/kalluri.html

All recent recommendations

• 3.2
  Asialoerythropoietin prevents contrast-induced nephropathy.
  Recommended by Raghu Kalluri with Keizo Kanasaki, Samuel Heyman
  
• 6.0
  Immigrating progenitor cells contribute to human podocyte turnover.
  Recommended by Raghu Kalluri with Dr Keizo Kanasaki
  
• 6.0
  Snail activation disrupts tissue homeostasis and induces fibrosis in the adult kidney.
  Recommended by Raghu Kalluri
  
• 3.2
  Smooth muscle alpha-actin deficiency in myofibroblasts leads to enhanced renal tissue fibrosis.
  Recommended by Jerome Rossert, Raghu Kalluri