Jack Satsangi

Gastrointestinal Unit, Western General Hospital, United Kingdom

Jack Satsangi, MD

Academic positions:
- Professor of Gastroenterology

All industry positions (in last five years):

Research interests:
Immunogenetics of inflammatory bowel disease

Any other information:

Any competing interests declared are displayed with individual evaluations.

Faculty Member: Gastroenterology & Hepatology > Inflammatory Bowel Disease (since 14 November 2005)

Links

http://www.cir.med.ed.ac.uk/

All recent recommendations

• 3.0
  Loss of the autophagy protein Atg16L1 enhances endotoxin-induced IL-1 production
  Recommended by Jack Satsangi with Charlie Lees
  
• 6.0
  A key role for autophagy and the autophagy gene ATG16L1 in mouse and human intestinal Paneth cells
  Recommended by Jack Satsangi with Charles Lees
  
• 3.0
  Sequence variants in IL10, ARPC2 and multiple other loci contribute to ulcerative colitis susceptibility.
  Recommended by Jack Satsangi
  
• 3.0
  Deletion polymorphism upstream of IRGM associated with altered IRGM expression and Crohn's disease
  Recommended by Jack Satsangi
  
• 3.0
  High frequency of early colorectal cancer in inflammatory bowel disease.
  Recommended by Jack Satsangi
  
• 3.0
  TL1A (TNFSF15) regulates the development of chronic colitis by modulating both T-helper 1 and T-helper 17 activation.
  Recommended by Jack Satsangi
  
• 3.0
  Replication of signals from recent studies of Crohn's disease identifies previously unknown disease loci for ulcerative colitis.
  Recommended by Jack Satsangi
  
• 3.0
  Natural History of Pediatric Crohn's Disease: A Population-Based Cohort Study.
  Recommended by Jack Satsangi
  
• 3.0
  Microbial influences in inflammatory bowel diseases.
  Recommended by Jack Satsangi
  
• 3.0
  The Paneth cell alpha-defensin deficiency of ileal Crohn's disease is linked to Wnt/Tcf-4.
  Recommended by Jack Satsangi
  
• 3.0
  IL-22 ameliorates intestinal inflammation in a mouse model of ulcerative colitis.
  Recommended by Jack Satsangi
  
• 6.0
  Unravelling the pathogenesis of inflammatory bowel disease.
  Recommended by Jack Satsangi
  
• 6.5
  A genome-wide association study identifies IL23R as an inflammatory bowel disease gene.
  Recommended by Jack Satsangi, Eduard Stange, Lars Rogge
  
• 3.0
  Defective acute inflammation in Crohn's disease: a clinical investigation.
  Recommended by Jack Satsangi
  
• 3.2
  Reduced Paneth cell alpha-defensins in ileal Crohn's disease.
  Recommended by Nur Alam, Jack Satsangi