Rahul Kuver

Gastroenterology, University of Washington, United States of America

Rahul P. Kuver M.D.

Academic positions:
- Associate Professor of Medicine, Division of Gastroenterology

All industry positions (in last five years):
None declared

Research interests:
Physiology of gallbladder and biliary epithelium, using techniques of cell culture, biochemistry, cellular and molecular biology. Relationship between CFTR and mucin secretion.

Functional Interactions between CFTR and the Ileal Bile Acid Transporter. Functional Interactions between CFTR and P-glycoprotein. Mechanisms of Cholesterol Transport in Gallbladder Epithelial Cells.

Any other information:

Any competing interests declared are displayed with individual evaluations.

Faculty Member: Gastroenterology & Hepatology > Biliary Tract (since 14 December 2006)

Links

http://www.uwgi.org/directory/results.asp?id=38

All recent recommendations

• 6.0
  Effect of ezetimibe on the prevention and dissolution of cholesterol gallstones.
  Recommended by Rahul Kuver
  
• 6.0
  Hepatic insulin resistance directly promotes formation of cholesterol gallstones.
  Recommended by Rahul Kuver
  
• 6.0
  Pathophysiological basis of liver disease in cystic fibrosis employing a {Delta}F508 mouse model.
  Recommended by Rahul Kuver
  
• 3.2
  Activation of liver X receptor sensitizes mice to gallbladder cholesterol crystallization.
  Recommended by Rahul Kuver, Cynthia Ko
  
• 4.8
  Immunoglobulin G4-associated cholangitis: clinical profile and response to therapy.
  Recommended by Rahul Kuver, Ulrich Beuers
  
• 6.0
  T-cell function is critical for murine cholesterol gallstone formation.
  Recommended by Rahul Kuver
  
• 6.0
  Direct intestinal cholesterol secretion contributes significantly to total fecal neutral sterol excretion in mice.
  Recommended by Rahul Kuver
  
• 9.0
  Increased gallstone risk in humans conferred by common variant of hepatic ATP-binding cassette transporter for cholesterol.
  Recommended by Rahul Kuver
  
• 3.0
  Bone marrow fails to differentiate into liver epithelium during murine development and regeneration.
  Recommended by Rahul Kuver
  
• 3.0
  MUC5AC, a gel-forming mucin accumulating in gallstone disease, is overproduced via an epidermal growth factor receptor pathway in the human gallbladder.
  Recommended by Rahul Kuver
  
• 6.0
  Identification of a hormonal basis for gallbladder filling.
  Recommended by Rahul Kuver